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170 Newest Publications about the topic traumatic brain injuries

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Comparison of methods for clustering independent EEG components in healthy subjects and patients with postconcussion syndrome after traumatic brain injury

24-Apr-2010 | V. A. Ponomarev, O. E. Gurskaya, Yu. D. Kropotov, L. V. Artjushkova, A. Müller, Human Physiology, 2010

The efficiencies of three clustering methods for independent components of 19-channel baseline EEG in location of pathological cerebral activity sources were compared. The samples comprised 518 healthy subjects and 87 patients with postconcussion syndrome after traumatic brain injury (TBI). ...

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Transient Receptor Potential Melastatin 2 Expression is Increased Following Experimental Traumatic Brain Injury in Rats

22-Mar-2010 | Naomi L. Cook, Robert Vink, Stephen C. Helps, Jim Manavis, Corinna van den Heuvel, Journal of Molecular Neuroscience, 2010

Traumatic brain injury (TBI) elicits a sequence of complex biochemical changes including oxidative stress, oedema, inflammation and excitotoxicity. These factors contribute to the high morbidity and mortality following TBI, although their underlying molecular mechanisms remain poorly understood. ...

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Traumatic brain injury and amyloid-β pathology: a link to Alzheimer's disease?

10-Mar-2010 | Victoria E. Johnson; William Stewart; Douglas H. Smith, Nature Reviews Neuroscience, 2010

Traumatic brain injury and amyloid-β pathology: a link to Alzheimer's disease? Nature Reviews Neuroscience 11, 361 (2010). doi:10.1038/nrn2808 Authors: Victoria E. Johnson, William Stewart & Douglas H. Smith Traumatic brain injury (TBI) has devastating acute effects and in many cases ...

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Changes in Brain Cholesterol Metabolome After Excitotoxicity

05-Feb-2010 | Wei-Yi Ong, Ji-Hyun Kim, Xin He, Peng Chen, Akhlaq A. Farooqui, Andrew M. Jenner, Molecular Neurobiology, 2010

Excitotoxicity due to excess stimulation of glutamate receptors in neurons is accompanied by increased Ca2+ influx, stimulation of Ca2+-dependent enzymes, ATP depletion, increase in lipid peroxidation products, and loss of glutathione. These changes resemble neurochemical alterations in acute ...

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Vascular endothelial growth factor increases neurogenesis after traumatic brain injury

13-Jan-2010 | Orli Thau-Zuchman, Esther Shohami, Alexander G Alexandrovich, Ronen R Leker, Journal of Cerebral Blood Flow & Metabolism, 2010

Vascular endothelial growth factor increases neurogenesis after traumatic brain injury Journal of Cerebral Blood Flow & Metabolism advance online publication, January 13, 2010. doi:10.1038/jcbfm.2009.271 Authors: Orli Thau-Zuchman, Esther Shohami, Alexander G ...

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Role of CCL2 (MCP-1) in traumatic brain injury (TBI): evidence from severe TBI patients and CCL2|[minus]||[sol]||[minus]| mice

23-Dec-2009 | Bridgette D Semple, Nicole Bye, Mario Rancan, Jenna M Ziebell, M Cristina Morganti-Kossmann, Journal of Cerebral Blood Flow & Metabolism, 2009

Role of CCL2 (MCP-1) in traumatic brain injury (TBI): evidence from severe TBI patients and CCL2−/− mice Journal of Cerebral Blood Flow & Metabolism 30, 769 (April 2010). doi:10.1038/jcbfm.2009.262 Authors: Bridgette D Semple, Nicole Bye, Mario Rancan, Jenna M ...

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Early nonischemic oxidative metabolic dysfunction leads to chronic brain atrophy in traumatic brain injury

23-Dec-2009 | Yueqiao Xu, David L McArthur, Jeffry R Alger, Maria Etchepare, David A Hovda, Thomas C Glenn, Sungcheng Huang, Ivo D ..., Journal of Cerebral Blood Flow & Metabolism, 2009

Early nonischemic oxidative metabolic dysfunction leads to chronic brain atrophy in traumatic brain injury Journal of Cerebral Blood Flow & Metabolism 30, 883 (April 2010). doi:10.1038/jcbfm.2009.263 Authors: Yueqiao Xu, David L McArthur, Jeffry R Alger, Maria Etchepare, ...

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Hypothermia Therapy for Brain Injury

10-Jul-2009 | Kenneth R. Diller; Liang Zhu, Annual Review of Biomedical Engineering, 2009

Induced hypothermia is an acknowledged useful therapy for treating conditions that lead to cell and tissue damage caused by ischemia, including traumatic brain injury, stroke, and cardiac arrest. An accumulating body of clinical evidence, together with several decades of research, has documented ...

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Post‐injury baicalein improves histological and functional outcomes and reduces inflammatory cytokines after experimental traumatic brain injury

01-Dec-2008 | Chen, S‐F; Hsu, C‐W; Huang, W‐H; Wang, J‐Y, British Journal of Pharmacology, 2008

Post-injury baicalein improves histological and functional outcomes and reduces inflammatory cytokines after experimental traumatic brain injury British Journal of Pharmacology 155, 1279 (December 2008). doi:10.1038/bjp.2008.345 Authors: S-F Chen, C-W Hsu, W-H Huang & J-Y Wang

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Nonlinear Assessment of Cerebral Autoregulation from Spontaneous Blood Pressure and Cerebral Blood Flow Fluctuations

15-Dec-2007 | Kun Hu, C. K. Peng, Marek Czosnyka, Peng Zhao, Vera Novak, Cardiovascular Engineering, 2007

Cerebral autoregulation (CA) is an most important mechanism responsible for the relatively constant blood flow supply to brain when cerebral perfusion pressure varies. Its assessment in nonacute cases has been relied on the quantification of the relationship between noninvasive beat-to-beat blood ...

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